Dear Readers, Welcome to Anti Adrenergic drug Objective Questions and Answers have been designed specially to get you acquainted with the nature of questions you may encounter during your Job interview for the subject of Anti Adrenergic drug Multiple choice Questions. These Objective type Anti Adrenergic drug Questions are very important for campus placement test and job interviews. As per my experience good interviewers hardly plan to ask any particular question during your Job interview and these model questions are asked in the online technical test and interview of many Medical Industry.
a) Prazosin
b) Phentolamine
c) Metoprolol
d) Reserpine
a) Phentolamine
b) Dihydroergotamine
c) Prazosin
d) Labetalol
a) Yohimbine
b) Tamsulosin
c)Tolazoline
d)Prazosin
a) Tolazoline
b) Labetalol
c) Prazosin
d) Phenoxybenzamine
a) Metoprolol
b) Atenolol
c) Propranolol
d) Acebutolol
a) Propranolol
b) Metoprolol
c) Carvedilol
d) Sotalol
a) Tolazolin
b) Pindolol
c) Ergotamin
d) Butoxamine
a) Propranolol
b) Metoprolol
c) Pindolol
d) Betaxolol
a) Labetalol
b) Phentolamine
c) Metoprolol
d) Propranolol
a) Tolazoline
b) Reserpine
c) Carvedilol
d) Prazosin
a) Reversible or irreversible interaction with adrenoreceptors
b) Depletion of the storage of catecholamines
c) Blockade of the amine reuptake pumps
d) Nonselective MAO inhibition
a) They cause a fall in peripheral resistance and blood pressure
b) They cause epinephrine reversal (convert a pressor response to a depressor response)
c) Bronchospasm
d) They may cause postural hypotension and reflex tachycardia
a) Prazosin
b) Labetalol
c) Phenoxybenzamine
d) Phentolamine
a) Reduction in peripheral resistance
b) Stimulation of responses to serotonin
c) Tachycardia
d) Stimulation of muscarinic, H1 and H2 histamine receptors
a) Antagonism of presynaptic alpha2 receptors enhances norepinephrine release, which causes cardiac stimulation via unblocked beta receptors
b) Baroreflex mechanism
c) Direct effect on the heart by stimulation of beta1 receptors
d) Inhibition of transmitter reuptake at noradrenergic synapses
a) Asthma
b) Cardiac arrhythmias
c) Pheochromocytoma
d) Chronic hypertension
a) Inhibition of the release of epinephrine from the adrenal medulla
b) Blockade of alpha2 receptors on vascular smooth muscle results in epinephrine stimulation of unblocked alpha2 receptors
c) Direct interaction with and inhibition of beta2 adrenoreceptors
d) Antagonism to the release of rennin
a) Phenylephrine
b) Propranolol
c) Phentolamine
d) Epinephrine
a) Selective beta2-receptor antagonists
b) Nonselective beta-receptor antagonists
c) Indirect-acting adrenoreceptor antagonist drugs
d) ?lpha-receptor antagonists
a) Diarrhea
b) Bradycardia
c) Arrhythmias
d) Myocardial ischemia
a) Ergotamine
b) Prazosin
c) Phenoxybenzamine
d) Carvedilol
a) Phentolamine
b) Phenoxybenzamine
c) Ergotamine
d) Prazosin
a) Irreversible blockade of alpha receptors
b) Highly selective for alpha1 receptors
c) The relative absence of tachycardia
d) Persistent block of alfa1 receptors
a) There are at least three subtypes of alfa1 receptors, designated alfa1a, alfa1b and alfa1d
b) ALPHA1a subtype mediates prostate smooth muscle contraction
c) ALPHA1b subtype mediates vascular smooth muscle contraction
d) ALPHA1a subtype mediates both vascular and prostate smooth muscle contraction
a) Prazosin
b) Tamsulosin
c) Phenoxybenzamine
d) Phentolamine
a) Hyperthyroidism
b) Cardiac arrhythmias
c) Benign prostatic hyperplasia (BPH)
d) Asthma
a) Phentolamine
b) Tolazoline
c) Ergotamine
d) Prazosin
a) Propranolol
b) Phentolamine
c) Tamsulosin
d) Ergotamine
a) The heart
b) The blood vessels
c) The renin-angiotensin system
d) All of the above
a) They occupy beta receptors and competitively reduce receptor occupancy by catecholamines or other beta agonists
b) They do not cause hypotension in individuals with normal blood pressure
c) They induce depression and depleted stores of catecholamines
d) They can cause blockade in the atrioventricular node
a) The negative inotropic and chronotropic effects
b) Acute effects of these drugs include a fall in peripheral resistance
c) Vasoconstriction
d) Reduction of the release of rennin
a) Increase plasma concentrations of HDL and decrease of VLDL
b) Bronchoconstriction
c) Decrease of aqueous humor prodaction
d) “membrane-stabilizing” action
a) Stimulation of lipolysis
b) Stimulation of gluconeogenesis
c) Inhibition of glycogenolysis
d) Stimulation of insulin secretion
a) It decreases cardiac work and oxygen demand
b) It reduces blood flow to the brain
c) It inhibits the renin secretion
d) It increases the atrioventricular nodal refractory period
a) Bronchoconstriction
b) “supersensitivity” of beta-adrenergic receptors (rapid withdrawal)
c) Hyperglycemia
d) Sedation, sleep disturbances, depression and sexual dysfunction
a) Cardiovascular diseases
b) Hyperthyroidism
c) Migraine headache
d) Bronchial asthma
a) Are members of the beta1-selective group
b) Are nonselective beta antagonists
c) Have intrinsic sympathomimetic activity
d) Have an anesthetic action
a) Propranolol
b) Metoprolol
c) Nadolol
d) Timolol
a) Metoprolol
b) Propranolol
c) Nadolol
d) Pindolol
a) Betaxolol
b) Sotalol
c) Nadolol
d) Metoprolol
a) Propranolol
b) Oxprenolol
c) Sotalol
d) Atenolol
a) Propranolol
b) Oxprenolol
c) Metoprolol
d) Carvedilol
a) They are nonselective beta antagonists
b) They have no partial agonist activity
c) They are less likely to cause bradycardia and abnormalities in plasma lipids
d) They are effective in hypertension and angina
a) Labetalol
b) Betaxolol
c) Propranolol
d) Timolol
a) It is a beta1 -selective antagonist
b) It has both alfa1-selective and beta-blocking effects
c) It attenuates oxygen free radical-initiated lipid peroxidation
d) It inhibits vascular smooth muscle mitogenesis
a) Prazosin
b) Propranolol
c) Reserpine
d) Phentolamine
ANSWERS:
1. b
2. C
3. a
4.d
5.c
6.b
7.d
8.c
9. a
10.b
11.a
12. c
13.d
14.b
15.a
16.c
17.b
18.c
19.d
20.b
21.a
22.b
23.a
24.d
25.b
26.c
27.d
28.b
29.d
30.c
31.b
32.a
33.c
34.b
35.c
36.d
37.a
38.b
39.c
40.a
41.c
42.b
43.b
44.a
45.a
46.c